Cell Engineering: Apoptosis (Cell Engineering) by Mohamed Al-Rubeai (Editor), Martin Fussenegger (Editor)

Cell Engineering: Apoptosis (Cell Engineering) by Mohamed Al-Rubeai (Editor), Martin Fussenegger (Editor)

By Mohamed Al-Rubeai (Editor), Martin Fussenegger (Editor)

The sequence mobilephone Engineering is the 1st and in basic terms significant reference paintings at the improvement of mobile structures for the creation of recombinant glycoproteins, gene and cellphone treatments, drug screening and tissue engineering. This quantity on "Apoptosis" is meant to check the cutting-edge with in-depth tests of this sort of programmed telephone dying. the purpose of the quantity is to make the hot advancements in apoptotic examine effortlessly available to biologists, biotechnologists and mobile engineers. The implication of apoptosis within the suppression of ailments and prolonging survival of cells in tradition is gifted to point the nice power of apoptotic examine for drug construction and the advance of human cures. All chapters are written as self-contained remedies of the real subject matters in apoptosis which are awarded on a vital details foundation. issues lined variety from knowing the position of signalling and effector molecules, mathematical modelling of mobile demise, RNAi instruments in apoptosis learn, to tracking and imaging of apoptosis. This quantity can be a useful source for biotechnologists and researchers in apoptosis, mobile biology, phone tradition and molecular medication.

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2002). Inducible expression of Bcl-XL restricts apoptosis resistance to the antibody secretion phase in hybridoma cultures. Bioeng. 79, 180-187. , and Harlan, J. M. (1996). Endothelial cell death induced by tumor necrosis factor-alpha is inhibited by the Bcl-2 family member, A1. Chem. 271, 27201-27204. Kerr, J. , Wyllie, A. , and Currie, A. R. (1972). Apoptosis: a basic biological phenomenon with wide-ranging implications in tissue kinetics. Cancer 26, 239-257. Khaled, A. , and Durum, S. K. (1999).

1999) NMR structure and mutagenesis of the inhibitor-of-apoptosis protein XIAP. Nature 401, 818-22. , Takio, K. and Takahashi, R. (2001) A serine protease, HtrA2, is released from the mitochondria and interacts with XIAP, inducing cell death. Cell 8, 613-21. Thornberry, N. A. and Lazebnik, Y. (1998. Caspases: enemies within. Science 281, 1312-6. Thornberry, N. , Rano, T. , Peterson, E. , Rasper, D. , Houtzager, V. , Nordstrom, P. , Vaillancourt, J. , Chapman, K. T. and Nicholson, D. W. (1997. A combinatorial approach defines specificities of members of the 22 H.

Green, D. R. and Reed, J. C. (1998). Mitochondria and apoptosis. Science 281, 1309-1312. Griffiths, G. , Morgan, C. , Jones, N. , Corfe, B. , and Hickman, J. A. (1999). Cell damage-induced conformational changes of the proapoptotic protein Bak in vivo precede the onset of apoptosis. Cell Biol. 144, 903-914. , Wei, M. , and Korsmeyer, S. J. (1998). Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis. EMBO J. 17, 38783885. , Yin, X. , Wei, M. , and Korsmeyer, S.

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