Textbook of Neuro-Oncology by Mitchel S. Berger, MD, and Michael D. Prados, MD (Eds.)
By Mitchel S. Berger, MD, and Michael D. Prados, MD (Eds.)
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Sample text
Part 1: Pathology. J Neurosurg 79:859–866, 1993. Porta-Etessam J, Berbel A, Martinez-Salio A, et al: Gliomatosis cerebri. MRI, SPECT and the study of pathology. Rev Neurol 29:287–288, 1999. Reni M, Ferreri AJ, Garancini MP, Villa E: Therapeutic management of primary central nervous system lymphoma in immunocompetent patients: results of a critical review of the literature. Ann Oncol 8:227–234, 1997. Rippe DJ, Boyko OB, Fuller GN, et al: Gadopentetate-dimeglumine-enhanced MR imaging of gliomatosis cerebri: appearance mimicking leptomeningeal tumor dissemination.
10,14, On imaging, they tend to have a well-circumscribed border, often composed of a solid enhanced nodule and a cystic component. Peritumoral edema is usually minimal, even in large lesions. PRIMARY BRAIN TUMORS OF NONGLIAL ORIGIN Primary Cerebral Lymphoma Over the past 2 decades, the incidence of primary cerebral lymphoma (PCL) has substantially increased in both immunocompromised and immune-competent individuals—a phenomenon that cannot be entirely explained by changes in tumor classification, the increased prevalence of AIDS, or the growing number of organ transplantations.
C, Axial fluid-attenuated inversion recovery image demonstrates extensive edema surrounding C the mass. pose a considerable diagnostic challenge to both the clinician and the radiologist. Clinically, TDLs may present with symptoms suggestive of a mass lesion. On MRI, TDLs are often indistinguishable from high-grade glial neoplasms, demonstrating ill-defined borders, mass effect, perilesional edema, central necrosis, contrast enhancement, and variable involvement of gray-matter structures. Even in the case of an established demyelinating disease such as multiple sclerosis, the atypical appearance of a large lesion should cause concern about a concurrent neoplasm.