MACHINING DEPLETED URANIUM by Hurst, J.S.; Read, A.M.; Union Carbide Corp., Oak Ridge,
By Hurst, J.S.; Read, A.M.; Union Carbide Corp., Oak Ridge, Tenn. Y-12 Plant; United States. Dept. of Energy.; Oak Ridge Y-12 Plant.; United States. Dept. of Energy. Office of Scientific and Technical Information
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5. Diagram showing the principal pathological processes following brain injury from the onset of BBB breakdown to the period when the BBB is restored. Morphology and Properties of Brain Endothelial Cells 23 an increased density of endothelial caveolae only in the vessels with BBB breakdown to HRP within minutes of the onset of pathological states such as hypertension, spinal cord injury, seizures, experimental autoimmune encephalomyelitis, excitotoxic brain damage, brain trauma, and BBB breakdown-induced by bradykinin, histamine, and leukotriene C4 (129, 182, 202) (Fig.
Tilling T, Korte D, Hoheisel D, Galla HJ (1998) Basement membrane proteins influence brain capillary endothelial barrier function in vitro. J Neurochem 71:1151–1157 35 82. Nusrat A, Parkos CA, Verkade P, Foley CS, Liang TW, Innis-Whitehouse W, Eastburn KK, Madara JL (2000) Tight junctions are membrane microdomains. J Cell Sci 113 (Pt 10): 1771–1781 83. Morita K, Furuse M, Fujimoto K, Tsukita S (1999) Claudin multigene family encoding four-transmembrane domain protein components of tight junction strands.
Peptide Transport Endothelial cells at the BBB express several transport systems for neuroactive peptides such as enkephalins (163), tyrosine melanocyte-stimulating inhibitory factor 1 (tyr-MIF-1), delta-sleep inducing peptide (164), luteinizing-hormone releasing hormone (LHRH), and some cytokines and chemokines (165, 166). The V1-vasopressinergic receptor is required for transport of argininevasopressin (AVP) from blood to the brain (167). Peptide transport systems 1 and 2 at the abluminal membrane mediate efflux of encephalins/Tyr-MIF-1 and AVP, respectively, from the brain to the blood.