Lippincott's Illustrated Q&A Review of Rubin's Pathology by Raphael Rubin, Emanuel Rubin, Bruce A. Fenderson, David S.
By Raphael Rubin, Emanuel Rubin, Bruce A. Fenderson, David S. Strayer
Lippincott's Illustrated Q&A Review of Rubin's Pathology, moment version bargains updated, clinically correct board-style questions-perfect for path assessment and board prep. nearly 1,000 multiple-choice questions with distinct solution causes disguise often established subject matters typically and systemic pathology. The e-book is seriously illustrated with photographs within the query or solution clarification.
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Extra resources for Lippincott's Illustrated Q&A Review of Rubin's Pathology (2nd Edition)
Sample text
NO also inhibits platelet aggregation and mediates the killing of bacteria and tumor cells by macrophages. Histamine (choice B), leukotrienes (choice C), and thromboxane A2 (choice E) stimulate the contraction of smooth muscle cells. Diagnosis: Acute myocardial infarction 41 The answer is D: Macrophages. Coal workers’ pneumoconiosis reflects the inhalation of carbon particles. The characteristic pulmonary lesions of simple coal worker’s pneumoconiosis include nonpalpable coal-dust macules and palpable coaldust nodules, both of which are typically multiple and scattered throughout the lung as 1- to 4-mm black foci.
The other choices are less likely to cause direct injury to the lung in a patient with pneumonia. Diagnosis: Bacterial pneumonia 20 The answer is D: a2-Macroglobulin. Proteolytic enzymes that are released by phagocytic cells during inflammation are regulated by a family of protease inhibitors, including α1-antitrypsin and α2-macroglobulin. These plasma-derived proteins inhibit plasmin-activated fibrinolysis and activation of the complement system and help protect against nonspecific tissue injury during acute inflammation.
Healing by primary intention occurs in wounds with closely apposed edges and minimal tissue loss. Such a wound requires only minimal cell proliferation and neovascularization to heal, and the result is a small scar. Healing by secondary intention occurs in a gouged wound, in which the edges are far apart and in which there is substantial tissue loss. This wound requires wound contraction, extensive cell proliferation, and neovascularization (granulation tissue) to heal. Granulation tissue is eventually resorbed and replaced by a large scar that is functionally and esthetically unsatisfactory.