Lewis's Child and Adolescent Psychiatry: A Comprehensive by Andres Martin, Fred R. Volkmar
By Andres Martin, Fred R. Volkmar
Established for fifteen years because the normal paintings within the box, Melvin Lewis's Child and Adolescent Psychiatry: A accomplished Textbook is now in its Fourth version. lower than the editorial path of Andrés Martin and Fred R. Volkmar—two of Dr. Lewis's colleagues on the world-renowned Yale baby research Center—this vintage textual content emphasizes the connection among uncomplicated technological know-how and medical learn and integrates medical ideas with the realities of drug interactions.
This variation has been reorganized right into a extra compact, clinically appropriate booklet and entirely up-to-date, with two-thirds new contributing authors. the hot constitution contains economics, range, and a heavy concentrate on evidence-based perform. a variety of new chapters contain genetics, examine method and data, and the continuum of care and location-specific interventions.
A significant other web site offers quick entry to the entire, totally searchable text.
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Additional info for Lewis's Child and Adolescent Psychiatry: A Comprehensive Textbook (4th Edition)
Example text
Can R Acad Sci 318:295–306, 1995. : A point mutation in the FMR-1 gene associated with fragile X mental retardation. Nat Genet 3:31–35, 1993. DeLorey TM, Handforth A, Anagnostaras SG, et al: Mice lacking the beta3 subunit of the GABA A receptor have the epilepsy phenotype and many of the behavioral characteristics of Angelman syndrome. J Neurosci 18:8505–8514, 1998. : doublecortin is the major gene causing X-linked subcortical laminar heterotopia (SCLH). Hum Mol Genet 7:1063–1070, 1998. : Short-term memory and cognitive variability in adult fragile X females.
In contrast, the lissencephalic brain lacks the normal pattern of sulci and gyri, and there are only four layers. (Adapted with permission from Reiner O, Lombroso PJ: Lissencephaly. ) In 1993, a large deletion on chromosome 17 was found in two patients with MDS. , 1993). This gene and genes on either side of it were absent owing to the deletion and is another example of a contiguous gene syndrome. The absence of several genes is believed to be responsible for the more severe MDS phenotype, which also has numerous congenital abnormalities.
Neuroreport 5:1509–1513, 1994. : Neuropsychological, neurological, and neuroanatomical profile of Williams syndrome. Am J Med Genet 6:115–125, 1990. Bird AP: Gene number, noise reduction and biological complexity. Trends Genet 11:94–100, 1995. Brannan CI, Bartolomei MS: Mechanisms of genomic imprinting. Curr Opin Genet Dev 9:164–170, 1999. : Fragile X syndrome: variability of phenotypic expression. J Amer Acad Child Adoles Psychiatry 4:463–471, 1987. : Triplet repeat mutations in human disease.